Hepatitis B causes liver cirrhosis

        Liver cirrhosis is a chronic disease with diffuse degeneration and necrosis of liver cells, liver fibrosis nodules widely. It is not an independent disease, but due to a variety of factors or injury or long-term repeated injury of hepatic cells, resulting in liver diffuse degeneration, necrosis and regeneration of liver, connective tissue diffuse hyperplasia, resulting in a pathological destruction of hepatic lobules and reconstruction, liver blood circulation disorders.

        Viral hepatitis cirrhosis caused by many reasons, the exact etiology is: suffering from viral hepatitis, especially hepatitis B and C can not get timely and sufficient rest, prematurely engaged in physical labor; the malnutrition, inadequate intake of protein and alcohol; the hepatitis B virus replication and proliferation of repeated treatment; not timely or not thorough; the illness during the recovery period or misuse of liver damage drugs, aggravated the liver damage; 6 with schistosomiasis. Chronic hepatitis can evolve into cirrhosis, severe hepatitis and caused extensive necrosis of liver cells can lead to necrosis after liver cirrhosis. So the development of chronic hepatitis B and C must pay great attention to.

        Chronic hepatitis inflammation to cirrhosis of varying lengths of time, short a few months, the elderly 10 to 20 years, and the condition is very different. Most of the patients with recurrent disease and the evolution of liver cirrhosis, some patients also 4-5 years had no obvious change, even the hepatitis B surface antigen (HBsAg) negative but for the development of liver cirrhosis.

        Hepatitis B cirrhosis clinically divided into active cirrhosis and cirrhosis still two kinds of active liver cirrhosis, chronic hepatitis B clinical manifestations include anorexia, abdominal distension, mild diarrhea, anemia, edema, male breast, female menstruation, splenomegaly, hypersplenism, liver palms, spider nevus; severe fever, jaundice, elevated transaminase, albumin decreased, liver texture changes, abdominal distension, lower extremity edema, ascites, portal hypertension, bleeding, liver failure, and even unconsciousness and coma. The rest of cirrhotic patients with normal transaminase, often without obvious jaundice, liver hard texture, splenomegaly, with portal hypertension, lower serum albumin.