Clinical findings and early signs of liver cirrhosis

        Cirrhosis most insidious onset, longer duration, and because the liver cells have strong compensatory function, so in the early hardening or even longer time, there are no obvious symptoms, and decompensation symptoms was clear, but the time has come a serious stage, prognosis.

        Clinical findings, in the course of cirrhosis, according to some of the symptoms, the discovery of occult cirrhosis of the liver has a certain significance.

        The chronic hemorrhage In liver cirrhosis, plasma coagulation factor decreases, heparin like material increases, blood platelet and capillary permeability increase, often can cause blood coagulation disorders and cause chronic bleeding. Such as repeated epistaxis, gingival bleeding, conjunctival blood stasis, subcutaneous bleeding, these findings may be early signs of liver disease.

The upper digestive tract hemorrhage

        The bleeding in liver disease in the previous years began to appear, and upper gastrointestinal bleeding is impaired in liver function and portal vein shunt, the gastric secretion cannot be inactivated, stomach caused by excessive secretion, an early sign or signal caused by acute erosive gastritis and acute ulcer. This is due to the stimulation of crude food, or acid and bile reflux corrosion, resulting in acute upper gastrointestinal bleeding. In addition, the blood flow of portal vein in liver cirrhosis is blocked, the pressure increased, esophageal and gastric varices rupture can cause bleeding. The upper digestive tract hemorrhage is a manifestation of hematemesis and melena.

- after infusion of ascites, edema

        This is caused by the loss of the function of the liver cell function and the portal vein system, the decrease of serum albumin and osmotic pressure. This is often in the treatment of other diseases, inappropriate excessive infusion, the results of ascites and edema. The infusion after the loss of appetite, often as a sign of obvious abdominal distension and early manifestation of the ascites.

The chronic cholecystitis, cholelithiasis

        Liver cirrhosis, gallbladder contraction, emptying slowly, self-cleaning effect is reduced, which is conducive to bacterial storage in the gallbladder, but also conducive to the precipitation of bilirubin and inflammatory exudate formation of stones. The clinical manifestations of right upper abdominal discomfort, jaundice, repeated indigestion, fever and so on.

        The chronic diarrheaThe portal venous reflux disorder, lead to intestinal mucosal edema, fat malabsorption and diarrhea, especially after eating fatty foods aggravate diarrhea.

The symptoms of estrogen increases

Liver compensatory function, capable of inactivating estrogen gradually weakened, resulting in increased estrogen, and the emergence of a series of symptoms, there is a common small vascular dilatation and local red cheeks, topless spider mole, palm and thenar and hypothenar redness, skin pigmentation, male impotence, female menstruation etc.. .

        One of the above symptoms, especially when several symptoms appear at the same time, should be alert to the occult cirrhosis of the signal, to do B ultrasound examination.

Many people are not easy to find their own liver cirrhosis, but the complications of gastric ulcer is relatively easy to find, so only pay attention to the treatment of gastric ulcer and ignore the treatment of cirrhosis, the delay of the disease.

        Medical experts found that patients with cirrhosis of the liver disease prone to ulcers. In some cases, it can be said that the ulcer disease is a clear signal of cirrhosis. They found that in a group of clinical observation in patients with liver cirrhosis patients with ulcer disease incidence was 15%, and the non cirrhosis canker on only 3.5%; a group of autopsy data showed that liver cirrhosis group ulcer disease incidence rate was 20%, while the control group was only 4% in liver cirrhosis.

        The secretion of gastric acid in patients with liver cirrhosis based most of the decrease, especially those with increased portal pressure in cirrhotic patients, often because of liver dysfunction, liver detoxification function, gastrointestinal mucosal circulatory disorders and hypoxia and other factors and damage of the gastric mucosal barrier, produce hydrogen ions reverse diffusion damage of gastric mucosa, and finally to the formation of ulcer disease.

        Therefore, when the loss of appetite, rhythm of upper abdominal pain, hematochezia and ulcer symptoms, or through the instrument check out time of ulcer disease, we must pay attention to whether there is cirrhosis.